((General post for the next two weeks or so. Preemptive tl;dr, find him anywhere, though suggestion of day/place/activity is appreciated
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i don't know /quits pretending to be organizedi_speak_softlyMay 7 2011, 16:55:45 UTC
[He swears you're also part-bloodhound.]
[Don hands Mike the book he nearly just dropped off the building.]
Decompression sickness (DCS) is due to the formation of inert gas bubbles in tissues and/or blood due to supersaturation, where either the mechanical stresses caused by bubbles or their secondary cellular effects cause organ dysfunction. DCS can be caused by a reduction in ambient pressure during ascent from a dive, rapid altitude excursion, in space or a hyperbaric/hypobaric chamber. Bubble formation occurs when decompression occurs sufficiently fast that tissue inert gas partial pressure exceeds ambient pressure, causing supersaturation and bubble formation. The resulting clinical manifestations include joint pains, cutaneous eruptions or rashes, neurological dysfunction, cardiorespiratory symptoms and pulmonary edema, shock and death. Several mechanisms have been hypothesized by which bubbles may exert their deleterious effects. These include direct mechanical disruption of tissue, occlusion of blood flow, platelet deposition and activation of the coagulation cascade, endothelial dysfunction and capillary leakage, complement activation and leukocyte-endothelial interaction.
[Don hands Mike the book he nearly just dropped off the building.]
Decompression sickness (DCS) is due to the formation of inert gas bubbles in tissues and/or blood due to supersaturation, where either the mechanical stresses caused by bubbles or their secondary cellular effects cause organ dysfunction. DCS can be caused by a reduction in ambient pressure during ascent from a dive, rapid altitude excursion, in space or a hyperbaric/hypobaric chamber. Bubble formation occurs when decompression occurs sufficiently fast that tissue inert gas partial pressure exceeds ambient pressure, causing supersaturation and bubble formation. The resulting clinical manifestations include joint pains, cutaneous eruptions or rashes, neurological dysfunction, cardiorespiratory symptoms and pulmonary edema, shock and death. Several mechanisms have been hypothesized by which bubbles may exert their deleterious effects. These include direct mechanical disruption of tissue, occlusion of blood flow, platelet deposition and activation of the coagulation cascade, endothelial dysfunction and capillary leakage, complement activation and leukocyte-endothelial interaction.
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Hm?
[Donnie Delayed Reaction.]
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I mean, are they CD or cartridge?
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They're little CDs.
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And the controllers? Oh, and the cords that came with it don't match anything.
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Thank you thank you thank you!
I'll make you some pie for dessert tonight! And... and cookies. And... [He's not going to offer to do your chores, Donnie. Sorry.]
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